Welcome User!
Case Study

Miasm & its Therapeutic Implication
NATIONAL JOURNAL OF HOMOEOPATHY 2003 May / Jun VOL V NO 3.
Dr Hitesh G Purohit
'Bac / Bac / Bac

(Editor: Miasm is all pervasive. Whether studying a remedy or disese or patient, the greater the clarity on the subject, the greater will be our efficacy in Homeopathic Practice.) 

The Hahnemannian classification of diseases based on theory of chronic miasms is a major breakthrough towards the rational therapeutics. Introduction of miasmatic classification of disease became the most important milestone not only for the Homoeopathic world but the entire medical fraternity. Unfortunately this unique contribution has become The most controversial in the Homoeopathic history. It has polarized the Homoeopathic profession. We have Allen, Kent & Robert on one side while Hughes, Hering & others stand on other side who have ignored, misunderstood or misinterpreted this doctrine. Allen is quite right in saying - the character of miasm yields the character of the disease or the form of the illness. Thus miasm - the residual poison in fact permeates into the cell, tissues & spirit of the individual and you see a distinct pattern in the evolution of disease. 

Masters have taught us that miasmatic concepts give an unique perspective to understand man in his totality

Each human being, right from the moment of conception, is characterized by a miasmatic modulation through which his individuality is constantly trying to emerge. Greater the miasmatic obstacles, deeper and extensive is its manifestations in health and disease. It is our day to day experiences of integration of miasmatic totality in handling of chronic illnesses. At the same time clinical experiences taught us immense value of using miasmatic force at a right time in ACUTE EMERGENCY CASES to prevent catastrophe.

Role of Miasmatic remedy in acute cases.
Bronchiolitis is known to be a paediatric emergency as it leads to complication rapidly.
Definition: It is an acute inflammation of the small airways resulting in a clinical syndrome characterized by inspiratory and expiratory wheezing and hyperinflation. More then 70 % cases are due to respiratory syncital virus (RSV)
Age: 2 to 9 months. Later occurrences fall under heading of Hypersensitive Airway Disorders.
Incidences: Mostly in winter & early spring, Males>Females, common in chubby children.
Risk Factors: babies not well breastfed. Crowded localities.H/O viral illnesses in family. Infants of smoking mothers

Clinical presentation: 
Patient presents with:
a) Abrupt respiratory distress with paroxysmal wheezy cough-a)dyspnoea, irritability & restlessness.
b) Non-acceptance of feeds-difficulty in sucking & swallowing.
c) Excessive crankiness.
d) Moaning.
On inquiry
a) H/o URTI in family with appetite diminished or mild fever.
b) Systemic manifestations like diarrhoea or vomiting may or may not be present.
Complications
Biochemical Changes.
a) Respiratory Failure
b) Pneumonia & its allied complications as it traverses from parenchyma to pleura to multiple organs, brain, blood kidney etc.

Physical Examination:
a) Look (sick/ill/active/playful)
b) alae nasi/use of accessory muscles.
c) moaning/grunting
d) Cyanosis - central/peripheral.
e) Temperature/resp rate.
f) On Auscultation - Fine crackles at the end of inspiration & early expiration. Prolonged expiration with
Audible wheezes. Liver and spleen palpable

Investigations:
a) CBC- Normal.
b) Chest X-Ray 1) Hyperluscent lung shadow.2) increase A.P. diameter of chest.3) flattening of ribs.- all signs of inflation

MANAGEMENT:
On the strength of respiratory distress and generals, Bronchiolitis is divided into mild, moderate & severe types. Mild to moderate can be managed at home. In severe Bronchiolitis, hospitalization will be required.
Miasm: Tubercular :
1) Sudden evolution & pace
2) Acute inflammatory response - oedema of bronchiolar wall, Hypersecretion of mucus, round cell infiltration, necrosis of cell wall & collection of cellular debris - Pathology of spasm more than exudate.
3) Low reactivity
4) Lingering infection.
The above understanding help us to handle serious disorder without many problems.

Case 1 :
Name: Master D Birth date: 23.8.99 (8 months)
D.O.C: 4.4.00.
Fa: 34. MO: 30.
Add. Vadodara.

Location

Sensation and Pathology

Modalities

Accompainments

RESP.SYSTEM
SINCE Jan.00. Every 15 days. Developed within 2 - 3 hrs.
Feb :00

RATTLING3, DYSPONEA3. Cough bouts2
Temp.100 - 101.
Diag: Pneumonitis.

<3change of atmos.<3 4 am
<3 lying down.. <Cold air,
>3 admission in Hospital for 4 - 8 days.treated with Steroids, nebuliser, antibiotics & bronchodilaters.
>Warm appl.

Weepy3 Clings to Mother.
Appetite Decreased.

Thermal State: Chilly.
Since last attack Pt.is on Beclate & Asthalin Inhaler twice a day.
O/E Chest: Nad.

Follow - Up:
Treatment was started with Bacillinum 200 every 15 days & inhaler was withdrawn within a week

Date

Sleep

Wee ping

Appe tie

N.D/
Rating

Whee zing

Dysp onea

RR/
Chest Temp.

Int / Exp

Action

13/5

N

- +

G

++

-

-

N

acute

Puls 200 bd

14/5

N

S

 

> / ++

+

_

26/
Ronchi +

 

Puls 200 4 hrly

15/5 am

Dis.

+++

Dis.

+++
10 Min

++

++

42/
Ronchi ++

Thirst 
++ Few, Freq.

Antim- ars 200 3 hrly

15/5 pm

S

> ?

S

> ?

Same

Once Inhaler

Ct all 2 hrly

16/5 am

S

s

s

s

s

S> inhaler

same

> then SQ

Bacillinum 200 Stat. Antim-ars 200 ct all

16/5 pm

> 2

> 2

> 2

32/ ronchi +

 

Antim-ars 200 4 hrly

17/5 pm

> 2

> 3

> 80 %

> 2

> 80

24/ ronchi +

 

Antim-ars 200 8 hrly

18/5

N

N

> 2

Occ

Occ

N

20/ Faint ronchi

 

 

Later the patient stabilised with weekly doses of the Intercurrent & Constitutional. Later on he had 3 more attacks in 2000 & one in 2001, which responded promptly to Bacillinum & Antim-ars 200 - 1m

CASE 2:
Master P: B.D.29.9.00 (6 months)
Fa: 30 yr. Mo: 26 yr. D.O.C:16.3.01.
Add: Vadodara.

Location

Sensation & Pathology

Modalities

Accompainments

Nose ----- chest 2 to 3 days.
Since 1st month of life.
Every month/remain for 5 to 6 days.
Since 14 Th c/o started

Running nose - watery - yellowish, greenish.
Noseblock2
Coughbouts2
Rattling3 - vomiting of curd like sticky profuse material
Breathlessness3
Temp 100 - 101
Diag :bronchiolitis

< 3 C.O.W.<2 Dust
< 2 evening
<2 Lying down, <3 2 - 4 am.
>2 vomiting >2 Vicks
>3 Nebuliser with Steroids & bronchodilaters. Antibiotics & bronchodilaters for 4 to 6 days.

Appetite 0. could not suck milk. Thirst Decreased.
Weepy2.Desire Mother at all times.
Carried desires to be
Eyes lachry : bland (Initially)

Thermal: C2H2 - C3H2.
F/H Fa: allergic bronchitis, Gr. Asthma. O/E Temp 100. Chest: Few creps with ronchi +. R.R 30 /min: Treatment Started with Pulsatilla 200 4 hourly.

Date

Mind

App
etie

Cough

Dysp
onea

Vomi
ting

RR/
Temp/Chest

Int / Exp
Other

Action

17/3/01

S

++
Sticky

30/99 - 100
Chest : s

Registration

Puls 200

18/3/01

S

S

~

S

Once

28/~/Chest
:Increased

SQ

Puls 200 3 hrly

19/3/01
9. am

S

S

~~
Mild >

> then
<

++ of sticky

35/99/
Chest:S

At Night :
Nebulisation
Disease ~
Inadequate
Response

Bacillinum 200 Stat. Puls 200 3 hrly

!9/3.PM

S

Once

32/99
Chest : >
Creps,
Ronchi S

Puls 200 3 hrly

20/3/01

> 2

> 2

> 2

0

28 / N /> 2

> 2

Puls 200 6 hrly

22/3/01

> 3

> 2

>80%

>3

0

N.Chest
:occ.ronchi

Puls 200 8 hrly

Later on Pt. stabilized with weekly doses of Calc-phos & Bacillinum 200 (infrequently).

Case 3: Name: Master M B D - 15.1.98.
D.O.C. 6.11.98. Address: Vadodara.

Location

Sensation & Pathology

Modalities

Accompaniments

Resp.system. Since 3 months of age.once/15 days.
Remain 5 - 6 days.
`Sudden within few hours.
Chest - Nose.
2nd day------

Rattling3,Cough Bouts3
Dysponea3 Vomiting
Thick3,White2
Sticky,Stringy2 Remove mechanically
Temp.101-103.
Diag :Bronchiolitis ----

<3C.O.W., <3Cold,
<2Banana,
<3 Evening, <2 1am.
>3 Hospitalized Treated with steroids,antibiotics & bronchodilaters(inj.)

Irritable2, Thirst Increased 2 few sips frequent.
Weakness2.

Thermal: Hot pt. F/H: Gf - Asthma, Pgmo: Eczema. O/e Chest: Nad.
4th & 5th Month developed Pneumonitis because of Bronchiolitis.
Treatment started with Calc-iod 200 wkly. In Nov - Last attack: pt. was serious, admitted & treated heavily with Inj.Steroid & Deriphyllin with Nebuliser Qds. For 3 days.

FOLLOW UP
6.12.98. _ Cold + Rattling 3 since Morning. Temp.100.Vomiting of food, was not able to sleep because of Cough.
O/E: Temp.100, Chest Ronchi +. Paed.consulted, who started Bronchodilaters + Steroids + Antibiotics. Ars-iod 200 6 hrly.

Date

Sleep

Mind

Appe tie

Cough/
Rattling

Dysp onea

Vomi ting

RR/
Temp.Chest

Int/
Exp

Action

7/12/98

Distur
bed

Irri
table

~~

+++

+

Twice

30/101
Chest - S

Ars-iod 200 4 hrly

8/12/98

S

S

S

++

4-5

34/100/S

Weak
ness

Ct all

8/12 pm

>Nap

s

s

s

>?

38/100/s

Neb
Once

Ars-iod 200 3 hrly

9/12/98

Dist

S

S

S

S

> 2

42/100/
Chest. Crep
Ronchi ++

X-ray:
Hyper
Inflation
Tc.11000

Bacillinum 200 stat.
Ars-iod 200 ct all

PM

S

> 2

>?

>2

S

36/100.
Chest>?

 

Ars-iod ct all

10/12

S

>2

> 2

0

26/N/
Chest >

 

Ars-iod 200 4 hrly

12/12/98

N

N

S

> 80 %

> 3

0

N

 

8 hrly

Later on Pt.settled with Calc-iod 200 with infrequent Intercurrent. He did develop attacks later on but responded promptly to Ars-iod 200 & Bacillinum.

CONCLUSION
All these cases beautifully demonstrate of need of Miasmatic remedy during Handling of acute serious disorder. Allen has mentioned "until you do not resolve miasmatic dyscrasia, you may not able to achieve rapid cure". Clarke in his introduction of Bacillinum write "Bad cases of asthma, Chronic respiratory disease, Copd, often helped by Bacillinum instead of other forces. Dr Cartier has written maximum on Bacillinum and has labeled Bacillinum a Syco- Tubercular remedy.

Biblography
1. Allen’s Chronic Miasm
2. Clarke & Cartier : Bacillinum as a drug.
3. Symposium Volume: ICR publication
4. I JHM volume on Nosodes
5. Paediatric in Homoeopathy : ICR publication