Welcome User!
Case Study

Breath Holding Spasms or Epilepsy?
NATIONAL JOURNAL OF HOMOEOPATHY 2003 Nov / Dec VOL V NO 6.
Dr. Hitesh Purohit
'Baryt-c

Master M Birth Date: 28/08/92 Hindu, Veg from Baroda
Fa 32 yrs, SYBCom. Tobacco business. Mo 27 yrs B.Com. Brother - 2yrs
Date of consultation: 11/04/96

Location

Sensation

Modality

Accompaniment

CNS
3rd month of life OD 2-6/d x 3-4 mins with occ gaps of 2 d
FACE to
CNS








Rectum since 1 week
10-15/ day
Abdome




H/O
Since 1st - 3rd yr of life
Rectum daily


3) MIND



Bluish Discoloration+
http://www.njhonline.com/images/downarrow.gif
Tonic and clonic spasm3
Foam occasional
http://www.njhonline.com/images/downarrow.gif
urinary incontinence
http://www.njhonline.com/images/downarrow.gif
p
 fall down.
o
 occ screams
sleepiness3
forget episode
Watery stools offensive3

Pain2
Mucus+
Involuntary+
Ineffectual urge2
Stool Report-NAD

Stool 4-5/day semi-solid. occ mucus.
Offensive. Involuntary.
Wt - maintained
Unusual behavior.
Aggressive
Beating3 Biting3
? MR? ADHD



A F Crying3
< fall down/ Injury
< Fasting2










< 3 Food after
not > Allop Rx x 1 wk
>3 stool after.





Not better by any medicine
No < modality
available

 

Physical Characteristics
Weight: 12 kg.
Perspiration: Partial: Head2, Palms- soles
Thirst-++11/2 glass, every ½hr.Desire for cold water3
Craving: Indigestible Things3, sweet3, ice cream2
Sleep: Startles during sleep. Fasting < 2
Thermal State: Chilly (C3H2)

Developmental Landmarks
Birth: FTND. Birth wt: 2.2kg. Crying: Immediately after birth
Mother’s Health: At 3rd month - Elder Son 4 yrs old, expired due to accident. Grief3. and now Anxiety 3 of this precious pregnancy. Low blood pressure, hyperemesis gravidarum for 3 months
Physical Development: 
Delayed. Wt gain- N
Mental Development: Delayed
Recognition-persons: 2 yrs of age
Teeth: 1 ¼ month. Sitting: 8 month. Walking: 1 ½ yr
Speech: Retarded http://www.njhonline.com/images/rtarrow.gif 3½ yr
Breast Feeding: 6 months
F/H
: Mother: convulsion-childhood during
O/E 
Nail-Pink Tongue- yellowish coated Wt-12 kg
CNS-NAD Neurologist. Dr K Buch opinion: Epilepsy-EEG: Abnormal.
Observation: 
Patient was restless and difficult to control by parents. Required time to answer simple questions.

Life-Space Investigation
This case was referred by family physician for c/o recurrent diarrheoa and convulsions. Parents were worried about recurrent convulsions.

Pt entered with parents in the consulting room. He appeared irritable, demanding chocolates and cold drinks. He did not listen to parents and started beating mother. Father gave Rs 100/- note to the driver and asked him to get whatever he wanted. I requested father to take him out for 30 minutes, which he readily agreed.

It is equally important to know about the family circumstances. They are belonging to Charoter Patel community. They have a family business of tobacco and supply raw tobacco to ITC. Pt’s grandfather is known as a tobacco king- a rich family. Pt’s father is the eldest son and generally remains busy3 traveling between Baroda http://www.njhonline.com/images/lftarrow.gif http://www.njhonline.com/images/rtarrow.gif Calcutta. During tobacco cultivation he stays out for months together.

Pt was the 2nd male child in family. 1st child expired (when pt was in womb). Mo passed through Anx3, grief3+. Pt was pampered3 / over protected3 by the family members. Grandfather fulfilled each and every demand, as if money is solution of all the problems! Later on father, as a compensatory mechanism, fulfilled demands. If demands were not fulfilled it resulted into crying http://www.njhonline.com/images/rtarrow.gif convulsion or burst out into anger, throwing and breaking things. It became a defense mechanism for the child to get whatever he wanted.

Gradually the child grew up in an environment where nobody scolded him. Later on mother noticed delayed development, and assistance in all routine activities. Even after repeated instructions he did not understand. Mother became worried when pt did not learn toilet training properly.

The child has been described as irritable3, loosing temper easily. When somebody comes home, at times he throws whatever things he has in his hand or grinds teeth. He behaves in the same manner (throwing things) while playing with other children. He talks to his elders in a loud voice and rough manner. He prefers to play alone; if plays with younger brother and his friends, it often ends in a fight.

At the same time he is a sensitive fellow, who misses his father and if somebody is ill in the family, he becomes calm and cool. Mother tried to put him in play group 2-3 times but in vain, as he used to fight with other children, did not listen to the teacher and used to break toys. Even private tuitions did not work out as he became rough and at times abusive to the teacher.

He is fond of roaming in car and often stops crying when the driver takes him for rides 3 to 4 times. Mother caught him for smoking bidi. Whenever scolded he responded by violence breaking T V/Radio/glasses etc.

Father is a cool and business3 oriented person. It appears that child does not have a fear of father; father does not have time for child.

Mother is a strict3 / disciplinarian3 lady who does try to improve child by various method but grand parents are a major block. Mother did not like grand parents’ behavior but Charotar Patel have no choice. When PP explained his parents about this likely diagnosis and that investigations were required, they doubted that best Pediatrician had never said about Epilepsy. I explained about the treatment and impact on development of the child then only they agreed for it.

My compunder said that his father had difficult time controlling the pt outside. A bribe of chocolates and soft drinks was given. Also passed urine in waiting room and soiled his underwear.

As a physician our first duty is to understanding pt at clinical level.
Either this child fall in category of
Infantile Spasms or Breath Holding Spells/Generalised Seizure.
It appears majority of Physicians took it likely diagnosed it as a breath holding spasm.
Criteria
1) Age of 6 month & 3 yr.
2) AF: crying/ppt factors: fear or minor injury.
3) Cyanosis then tonic, clonic spasm
4) If hypoxia persist more then 15 seconds then convulsion may occur.
5) Other phases of convulsions are absent.

Following data was available from the case:
Pt presented with symptoms of ictal and post ictal phase. Attacks also present when child was alone. Symptoms + incontinence of urine, falling on ground and post ictal sleepiness favour Epilepsy.

History of mother: suffered from epilepsy in childhood suggestive of genetic transmission.

I took help of neurophhysician (Dr. K.R.B) & he wrote, 15/4/96
Dear Dr Purohit,Thanks.Master M.P has severe epilepsy-confirmed on EEG.
I advised to put him on syp.Tegratol. With regards. Dr. K.R.B

EEG report 
of 15/04/96
Sedated EEG record is abnormal: Consistent with inter-ictal record of generalized seizures.
How do we understand his behavioral pattern?
 Either it is normal in pampered child or it is abnormal. Psychologist report helped to understand pt better.

Psychological Test Report
Master MP M/ 4.6yrs. Education: Nursery
Tests Administered: VSMS, BKT, Goddard form Board
Observation and Test Report: Patient was brought for assessment by his parents with h/o seizure? Breath Holding Spasms-on and of only when he cries. EEG also supports generalized seizures.
During both testing sessions, Patient was very restless, hyperactive, destructive and very attention seeking.

On Social-Developmental scale, his social maturity level is at 3 years level. He is unaware of any obstacles, shows no fear, and is partially dependent on mother for most ADL activities like toileting, dressing, bathing. He can learn given an opportunity, but he is not given a chance either because he is overprotected or that he is distracted and impulsive.
SQ=80 points-Dull Average Range
On BKT Tests of Verbal Abilities -his Mental Age=3 years-Verbal JQ being 72 Points- Dull Range:
His level of verbal comprehension is good, he can grasp easily but due to hyperactive behavior, his performance is affected. He can identify object body parts very well. His picture vocabulary and matching, recognition is good but it’s just that he fails to benefit from any learning due to distracted behavior. He has good concept in shapes and a few colours.
He is still at a scribbling stage. His adaptive functions are poor; probably diffuse brain pathology because of epilepsy could then manifest attention deficit disorder.

Superseding that over protection, overindulgence by parents and grandparents have probably spoilt child’s habits such that mother cannot control his stubborn behavior. Parents are very anxious and apprehensive about his fits, hence they fail to control his behavior and fulfill all his demands and tolerate all his mischief.
Father keeps away from child for days together on business trip and tries to compensate by over-pampering, when returns.
Mother tries to be overprotective vs strict disciplinarian yet cannot control him because of indulgence from grand parents.
Both parents are counseled. They feel putting him in a hostel might be a better solution. That should be their decision, may be later on

Diagnosis
: Dull average Intelligence (75to 80 point) with Epilepsy and ADHD (Attention Deficit Hyperactivity Disorder) Normal IQ range = 90+ points.
Recommendation: medication, parental counseling, regular schooling, re-evaluation every 6 months.
Once the understanding of the patient is clear at clinical level, then our job is easy & we can plan out homoeopathic management without difficulty.

Repertorial Totaly. Kent Approach

  1. Convulsion crying from: AF &<. Crying: Synthesis 205
  2. < alone: Synthesis 30
  3. Irritablity: violent, rage: Synthesis 202
  4. Learning with difficulty: btp 52
  5. Foolish, childish behaviour: Synthesis 110
  6. Obstinate: Synthesis 154
  7. Delayed development: Synthesis 1580
  8. < fasting
  9. Cr: indigestable
  10. Cr: sweet
  11. Cr ice-cream

Following drugs emerge from reportorial filter. Calc-carb, Baryta-carb, Nat-mur, Tarentula. 

Calc-c
 
comes near but basically calcarea child is capable of doing work but his sluggishness at level of mental & physical prevents him to do work.

Nat
 & Tarentula comes near at level of expression like irritability, destruction, violence etc & obstinacy but both these remedies do not cover the core of pt.

Baryta-carb:
 identification usually starts with delayed development or certain syphilitic expression present since birth. As the child grows up, shyness, non-cooperativeness, sluggishness and obstinacy develops because of dullness. As our pt grew up, he faced difficulty in school due to poor perception 
http://www.njhonline.com/images/rtarrow.gif shyness/anxiety or frustration expressed by irritability and violence. Our pt’s journey really began from fetal life where elder brother died due to accident resulting in a tremendously anxious state of mother http://www.njhonline.com/images/rtarrow.gif over-protection and over-pampering. So the second defect was in poor initial training of pt. All demands were fulfilled http://www.njhonline.com/images/rtarrow.gif Obstinacy. Development delayed and Dullness-school tuitions became essential. High degree of Freedom http://www.njhonline.com/images/rtarrow.gif Strictness resulted in difficulty in Adaptations, Low Tolerance http://www.njhonline.com/images/rtarrow.gif Aggresiveness, http://www.njhonline.com/images/rtarrow.gif Violence, http://www.njhonline.com/images/rtarrow.gif andInternal Restlessness
This understanding at level of mind + Body helped to come to. Baryta-carb

Intercurrent Remedy:
 Tuberculin bovinum:
Tuberculinum
: Seizure is basically an expression of aberrant electrical discharges. viz, hyper- excitability and erraticity. Genetic H/O mother also spells Tubercular miasm. Same time expression level of mind speaks of Tub-syphilitic zone.

Follow Up Criteria:

 

A]

 

 

 

1] Epilepsy A/F crying, Alone

6] Tonic / clonic spasms

 

 

2] Intensity

7] Involuntary urine

 

 

3] Frequency

8] Falling on ground

 

 

4] Duration

9] Sleepiness

 

 

5] Ictal phase cyanosis

 

 

 

B]

 

 

 

1] Involuntary stool

5] Mind in gen / parents behavior

 

 

2] Irritability

6] Diet/app/wt

 

 

3] Obstinacy

7] EEG

 

 

4] Abnormal development

8] IQ

 

Date

1

2

3

4

5

6

7

8

9

OBS/IN

Remedy

11/4/96

                 

Acute gastritis

Nux-v 200 4 hrly

15/4/96

                 

Acute over

Baryta-c 200

22/4/96
A]

S

S

S

S

S

S

Plays with
children

Baryta-c 200

B]

S

2

2

2

2

 

 

 

 

 

 

2/5/96
A]

S

<X2D
then
>

er-
ratic

S

S

S

S

S

Inadequate
control.
Freq rep + IR
requirements

Tub-b 1M
Baryta-c 200 3P/HS

B]

S

               

26th - 31st 5-6
times attack

 

9/5/96

S1>

S1>

S

S

S

?

0

Freq. repetition
All the time
crying No <

Baryta-c 200 2P

B]

S

               

Cr:Indigestible

 

16/5/96
B]

S
0

L7

L7

S

0

Cr:indigest3

Baryta-c 200 7 HS

31/5/96

B]

>/0

S

>

++

>

?

>

?

 

S

S

0

0

Attacks after
fighting. 2/2W.
Mind same

Baryta-c 200 BD

5/6/96

                 

Loose stool

Nux-v 200 4hrly
Baryta-c ct all

12/6/96

+++

               

overstimulation

SL

24/6/96
B]

>
?

>
?

>
?

>
less

S
>

S

S

0

0

< passed of

Baryta-c 1M/ 1D

8/7/96
B]

>
>2

1/5D

S

>/>
14

0

0

> 

Baryta-c 1M 1D

30/7/96
b]

0/0

>2

>2

>2
>

S

S

S/S

0

0

No attack since
5 days

Ct all

16/8/96
B]

0/0
>2

>2
?

>2
>2

>2
>

S
>

S/S

0

0

Initial 2 d
attack. then no
attack for 15 D.
no problem in
school

Tub-b 1M 1D
Baryta-c 1M 7HS

22/8/96

++

 

 

 

 

 

 

 

 

Med <

SL

26/8/96
B]

 

 

 

>3

>3

 

 

 

http://www.njhonline.com/images/rtarrow.gif >but disease activity constant

Baryta-c 1M 1D wkly

9/10/96
B]

>2
>3

>2+
>

>2+
>

>2+
>

0

>2+

0

0

0

 

Ct all x 2wks

2/11/96
B]

S


0

3/2W
>3

>
OK

>/0
>2

L5

0

0

0

Plays with other
children.Good 
Control

Ct all x 3wks

27/12/96

 

 

 

 

 

 

 

 

 

Irregular drugs.
Attacks ++.
Requires I.R

Tub-b 1M 1D
Baryta-c 1M 1D

11/1/97
B]

+/0
0

>2+


3/

>2+

0
15

0

0

0

 

Good Control

Baryta-c 1M 3p x 4wks

12/2/97

 

 

15D

 

 

 

 

 

 

Attacks>2+

Baryta-c 1M 1p x 4wks

27/2/97

 

 


3/2

 

 

 

 

 

 

Good Control.
needs freq repn

Baryta-c 1M 3P

13/3/97

++

 

wks
++

 

 

 

 

 

 

Need of I.R

Tub-b 1M 1D
Baryta-c 1M 3P

5/4/97

 

 

 

 

 

 

 

 

 

Acute GI with
fever.Acute followed
by C

NV 200 4 hrly http://www.njhonline.com/images/rtarrow.gif
Tub-b 1M http://www.njhonline.com/images/rtarrow.gif
Baryta-c 1M 3P

27/5/97

 

 

 

 

 

 

 

 

 

Out of Station.
Acute infection.

NV 200 4 hrly
Tub-b 1M
Baryta-c 1M3P x 3wks

7/6/97

++


>

 

 

 

 

 

CR req more
freq

Tub-b 1M
Baryta-c 1M 3P

17/10/97

 

 

 

 

 

 

 

 

 

Good control
Mind at rest

Tub-b 1M
Baryta-c 1M 1D

26/11/97

 

 

 

 

 

 

 

 

 

No attacks

Tub-b 1M
Baryta-c 1M 1D
week x 2wks

15/12/97

 

 

 

 

 

 

 

 

 

EEG>
Attack once

Baryta-c 1M 1D
week x 2wks

2/1/98

 

 

 

 

 

 

 

 

 

due to fall

Tub-b 1M http://www.njhonline.com/images/rtarrow.gif
Baryta-c 1M 1D

20/1/98

 

 

 

 

 

 

 

 

 

Good control

Baryta-c 1M 1P

25/3/98
B]

Mild
0

Mild
+

Twice
N

1/2
N

0
>

+

0

0

0

Good control
Skin+

Baryta-c 1M 1D

7/4/98

 

 

4/w

 

0

+

0

0

0

Good control
but ppt factor+
skin++

Psor 1M 1D
Baryta-c 1M 1D

21/4/98

 

 

 

 

 

 

 

 

 

Skin >3

Baryta-c 1M 3P

20/6/98

 

 

 

 

 

 

 

 

 

No attacks
Passed

Baryta-c 1M 1D

 

 

 

 

 

 

 

 

 

 

entrance exam
in Panchagani

 

17/7/98

 

 

 

 

 

 

 

 

 

Satbility

Baryta-c 1M 1D

 

Child now under observation only

 

 

                             

 

EEG report of 15/12/97
Awake EEG record is minimally abnormal- to be clinically correlated.
Present EEG shows less epigenocity as compare to EEG done on 15/04/96. EEG report: 20/06/98
Awake EEG is within normal limits.

Learning From The Case:
Management General Environment-Individual:- Initially problem started with clinical diagnosis. Physician should have a clarity what he is treating. And how cure is going to take place in this individual case. The way paediatrician took it loosely about infantile spasm tested physician’s knowledge and confidence. Sensitive handling of parents about clinical diagnosis removed agitation from their mind. Involving neurophysician at right juncture sorted out problems. Physician should not have any restriction in taking help from other sciences when better facilities are available. All "Specialist" should be used with "respect and care". They become asset if properly used.


Cure is only possible when we totally abolish disease process, while equally taking care of precipatating and maintaining factors. In this case family environment was playing a big role, crying used to <. Crying, mostly, become a defense mechanism for the child to fulfill all his demands. Overindulgence of grandparents and father lead to spoiling the child. If this environment was not corrected then chances of improvement was remote. They were explained about impact of over protectiveness/ pampering on development of the child. Further enviornment of child on intellectual and behaviour level was done by psychologist. Involving psychologist in process of cure was of an asset. Psychologist plays important role in assesment of pt and hence clear cut diagnosis were avilable. Same time she was able to give feedback to parents about their handling in a precise manner. All this provided, crystal clear clarity to the physician about his role not only limited to treatment of epilepsy but to change child’s adaptive-reactive pattern so he can adjust well and life may become more meaningful.

This type of cure is only possible when clinician perceive pt as a whole and does not treat parts.

Frequent repetition of medium potency was administred to control neuronal discharge (neuronal excitability with heightened sensitivity). Miasmatic care was taken in terms of infrequnent to frequent repitition of Tuberculinum. Gradually response started in both signs and symptoms and improvement found in EEG. Later on both came normal within stipulated time. General derivation occurred in form of use of medium to higher potency in frequent repitition, which is essential for rapid and permenent cure. Timely used of intercurrent remedy proves an asset in rapid resolution of case.

Slow but steady improvement occurred- first in behavioural pattern and later on intellectual pattern. Pt adjusted well and lastly he was accepted in boarding school at Panchgini. Pt was confident in acceptance of new environment yet further clarity will come later on as how he keeps balance in demand à capacity à expectation with limited resources.

Importance of detail follow ups, repeated investigation were advised to understand role of IR+CR. Frequent repitition of CR was used to control erraticity and hyper excitablility of mind and neurons. Once potency exhaused à next potency used and result achieved. Importance of Tuberculinum as IR was felt. Further recovery only occurred after intoducing IR at the right time. During follow-up also, whenever frequency was decreased, vigilance was maintained and SOS advice was given. (It was felt that sending out from environment might work as a resolution, so once pt has improved, green signal was given by the physician.

Reference Readings:
Books on pediatrics: Nelson, Forfar & OP Ghai.

Books on Neurology: Adams & Victor.
Principle & Practice: Dr ML Dhawale
Symosium Volume F & G: ICR Publication